Are a novel class of androgen receptor ligands. They are intended to have the same kind of effects as androgenic drugs but be much more selective in their action, allowing them to be used for more uses than the relatively limited legitimate uses of anabolic steroids.
YK11 was discovered in 2011 by Yuichiro Kanno of Toho University. It is a steroidal selective androgen receptor modulator (sarm). It is a gene-selective partial agonist of the androgen receptor
Sarms UP Myo Booster -YK11 is actually a myostatin inhibitor. Myostatin inhibits muscle growth. So by inhibiting the inhibitor, YK11 should elicit more pronounced muscle growth and has also been shown to be more anabolic than dihydrotestosterone (DHT).
Is YK-11 a Steroid or a SARM?
YK-11 was discovered When Yuichiro Kanno discovered YK-11, he dubbed it as a potential anabolic SARM. However, that label is a little misleading because it certainly has more in common with a steroid than a SARM.
One could theorize that the creators of this compound advocated it be referenced as a myostatin inhibiting SARM. Due to the social stigma attached to AAS, continued research funding for YK11 would be unlikely if labeled a steroid.
YK11 has a very similar chemical structure to Testosterone and DHT. This would explain why YK-11 has been shown to be more anabolic than dihydrotestosterone (DHT).
This compound does not have a 24 hour half-life like most sarms. And it is my understanding that it will last in your system around 8 to 10 hours.
Client have made the following comments in relation to Sarms Up Myo Booster YK11
“I got amazing strength gains after about a week of taking this stuff! The best way I could describe it was that I felt I had a certain command of heavier weights. I was banging out more reps on heavy weights and felt confident every time I decided to add weight to my maximums.”
“I really think that most of the gains I got probably came from my beloved RAD-140 but there is no doubt that I saw some nice changes in body composition due to being able to punch more weight.”
“The most noticeable thing though was how easily I could get a pump. I’m not exaggerating here when I say that I could just do some hard flexing to get a muscle group engorged with blood. So I’m sure that blood and nutrients getting to the various muscle groups easier contributed to my awesome experience here.”
YK11 is a new synthetic steroid based on 5-α-dihydrotestosterone (DHT), a hormone naturally found in the human body. DHT is a stronger form of testosterone that targets androgen receptors in the prostate, sex organs, hair, and liver.
YK11 is gaining interest from bodybuilders, who believe it can help them quickly build muscle with minimal side effects—a belief which is not founded in the available science. The YK molecule has more in common with steroids than SARMs, although it’s often mistakenly labeled as a non-steroidal SARM .
According to preliminary cell studies, YK11 may increase muscle mass and enhance bone health.
Some bodybuilders who have experimented with YK11 report great muscle gains and fat loss with minimal side effects.
- May increases muscle growth
- May boost fat-burning
- May increase bone strength
The chemical structure of YK11 is similar to DHT, and it binds to androgen receptors in a similar way. DHT is a naturally occurring hormone in the body crucial for hair growth, prostate health, and proper development in puberty.
Some bodybuilding resources state that YK11 is one of the strongest SARMs on the market. However, research revealed that YK11 only partially activates androgen receptors. Such limited activation increases the activity of catabolic (muscle-degrading) genes .
A recent cell study uncovered YK’s unique muscle-building mechanism: increasing follistatin levels. Follistatin is a naturally occurring protein that suppresses myostatin, which otherwise prevents muscles from getting too large. In comparison, no other SARMs show myostatin inhibition .YK11 may also increase bone growth by affecting a DHT-like pathway, according to other cellular studies. However, it is unclear whether this mechanism would transfer to living animal or human systems